Matt Ramer

Associate Professor
Department of Zoology/Neurosurgery

Phone: 604 675-8821

Web pages:
Lab webpage
Directory Listing



  • BSc Queen's University, Life Science, 1995
  • PhD Queen's University, Physiology, 1998


  • Spinal cord injury

Research Interests

My laboratory is interested in primary sensory nerve cells (neurons), which are responsible for the transmission of somatic (bodily) sensations such as touch, pain, hot, cold and so on from the periphery (skin, muscles and viscera) to the central nervous system (CNS, spinal cord and brain). Our major research focus is on changes that occur in sensory neurons and other cells of the nervous system when they are injured, the consequences to the physiology and behaviour of animals (rats and mice) with damaged sensory neurons, and ways to improve the outcome of such damage.

Selected Publications

Gaudet AD, et al. A role for galectin-1 in the immune response to peripheral nerve injury. Exp. Neurol. 220(2) 320-7

Inskip JA, et al. Cardiometabolic risk factors in experimental spinal cord injury. J. Neurotrauma

Inskip JA, et al. Autonomic assessment of animals with spinal cord injury: tools, techniques and translation. Spinal Cord 47(1) 2-35

Ramer MS. Anatomical and functional characterization of neuropil in the gracile fasciculus. J. Comp Neurol. 510(3) 283-96

Soril LJ, et al. Spinal brain-derived neurotrophic factor governs neuroplasticity and recovery from cold-hypersensitivity following dorsal rhizotomy. Pain 138(1) 98-110

Hampton DW, et al. A potential role for bone morphogenetic protein signalling in glial cell fate determination following adult central nervous system injury in vivo. Eur J Neurosci. 26(11) 3024-35

Gaudet AD, Ramer LM. Mind the GAP: a role for neurofibromin in restricting axonal plasticity. J Neurosci. 27(21) 5533-4

Ramer LM, et al. Endogenous TrkB ligands suppress functional mechanosensory plasticity in the deafferented spinal cord. J. Neurosci. 27(21) 5812-22